Innovative Research Award

Sara Amjad
Aligarh Muslim University, India

Sara Amjad
Affiliation	Aligarh Muslim University
Country	India
Scopus ID	57222341800
Documents	2
Citations	483
h-index	2
Subject Area	Computer Science
Event	Computer Scientists Awards
Google Scholar ID	MCnRe20AAAAJ&hl

Sara Amjad is a researcher associated with Aligarh Muslim University whose scholarly record includes work spanning cellular metabolism, molecular signaling, genetics, neuroscience, and computationally relevant biomedical research. Her documented publications include collaborative review research examining NAD⁺-dependent cellular processes and the genetics of glutamate signaling in autism spectrum disorder. These works provide evidence of interdisciplinary engagement across molecular biology, health sciences, and data-informed research.

Abstract

Sara Amjad’s research profile is represented by collaborative publications addressing biological signaling, metabolism, genetics, and neurodevelopment. Her work on NAD⁺ reviews its importance in energy metabolism, DNA repair, gene expression, cellular stress, aging, cancer, and neurodegeneration. [1] A second publication examines genetic alterations involving glutamate and its receptors in autism spectrum disorder, connecting molecular genetics with neuroscience and non-invasive imaging. [2]

Keywords

• Computer Science
• NAD⁺ metabolism
• Cellular signaling
• Genetics
• Neuroscience
• Autism spectrum disorder

Introduction

The supplied publication record indicates an interdisciplinary research orientation. In the NAD⁺ review, the authors describe NAD⁺ as a critical coenzyme involved in bioenergetics and cellular signaling, while discussing its relationship with aging, metabolic disorders, cancer, and neurodegeneration. [1] The autism spectrum disorder review evaluates genetic changes affecting glutamate and glutamate receptors and considers imaging approaches for investigating altered glutamatergic pathways. [2]

Research Profile

The provided Scopus information lists two documents, 483 citations, and an h-index of 2. These supplied metrics should be interpreted as a profile snapshot because citation databases can change over time. The publications also demonstrate collaboration across institutions and disciplines, including molecular medicine, genetics, neuroscience, imaging, and biomedical research.

Research Contributions

Amjad’s documented contribution includes participation in research that synthesizes evidence across complex biological systems. The NAD⁺ article identifies cellular energy metabolism, DNA damage repair, gene expression, and stress response as major areas influenced by NAD⁺ biology. [1] The genetics review addresses glutamatergic signaling and receptor biology in relation to autism spectrum disorder, emphasizing interactions among genetics, neuronal function, and imaging. [2]

Publications

• Role of NAD⁺ in regulating cellular and metabolic signaling pathways, Molecular Metabolism, 2021, 49:101195.. [1]
• Genetics of glutamate and its receptors in autism spectrum disorder, Molecular Psychiatry, 2022, 27:2380–2392. . [2]

Research Impact

The supplied citation count of 483 indicates that the documented publication set has received substantial scholarly attention relative to its size. The cited research addresses topics with broad biomedical relevance, including metabolism, cellular signaling, genetics, and neurodevelopment. The Nature publication also records citation and access metrics on its publisher page, illustrating continuing visibility of the research area. [2]

Award Suitability

Based on the supplied profile and publication evidence, Sara Amjad can be considered for an Innovative Research Award in recognition of interdisciplinary scholarly participation and research addressing contemporary questions in cellular metabolism, molecular genetics, neuroscience, and biomedical science. The assessment should remain subject to the award committee’s independent verification of affiliation, authorship, bibliographic records, and current research metrics.

Conclusion

Sara Amjad’s documented research contributions demonstrate engagement with interdisciplinary biomedical questions and collaborative scientific scholarship. Her publications on NAD⁺ signaling and glutamate genetics provide a foundation for evaluating her research profile within an innovation-focused academic recognition framework.

External Links

• Google Scholar Profile .
• Scopus Author Profile
• DOI
• Computer Scientists Awards

References

1. Amjad, S. et al. (2021). Role of NAD⁺ in regulating cellular and metabolic signaling pathways. Molecular Metabolism, 49, 101195.
   https://doi.org/10.1016/j.molmet.2021.101195
2. Nisar, S. et al. (2022). Genetics of glutamate and its receptors in autism spectrum disorder. Molecular Psychiatry, 27, 2380–2392.
   https://doi.org/10.1038/s41380-022-01506-w
3. PubMed. (2021). Role of NAD⁺ in regulating cellular and metabolic signaling pathways. PMID 33609766.
   https://pubmed.ncbi.nlm.nih.gov/33609766/
4. PubMed. (2022). Genetics of glutamate and its receptors in autism spectrum disorder. PMID 35296811.
   https://pubmed.ncbi.nlm.nih.gov/35296811/
5. Elsevier. (n.d.). Role of NAD⁺ in regulating cellular and metabolic signaling pathways. Molecular Metabolism.
   https://www.sciencedirect.com/science/article/pii/S2212877821000351
6. Springer Nature. (2022). Genetics of glutamate and its receptors in autism spectrum disorder. Molecular Psychiatry.
   https://www.nature.com/articles/s41380-022-01506-w